| Reversing EphB2 depletion rescues cognitive functions in Alzheimer model M Cissé, B Halabisky, J Harris, N Devidze, DB Dubal, B Sun, A Orr, G Lotz, ... Nature 469 (7328), 47-52, 2010 | 487 | 2010 |
| Phospholipase A2 reduction ameliorates cognitive deficits in a mouse model of Alzheimer's disease RO Sanchez-Mejia, JW Newman, S Toh, GQ Yu, Y Zhou, B Halabisky, ... Nature neuroscience 11 (11), 1311-1318, 2008 | 385 | 2008 |
| Neprilysin overexpression inhibits plaque formation but fails to reduce pathogenic Aβ oligomers and associated cognitive deficits in human amyloid precursor protein transgenic mice WJ Meilandt, M Cisse, K Ho, T Wu, LA Esposito, K Scearce-Levie, ... The Journal of Neuroscience 29 (7), 1977-1986, 2009 | 186 | 2009 |
| The disintegrin ADAM9 indirectly contributes to the physiological processing of cellular prion by modulating ADAM10 activity MA Cissé, C Sunyach, S Lefranc-Jullien, R Postina, B Vincent, F Checler Journal of Biological Chemistry 280 (49), 40624-40631, 2005 | 156 | 2005 |
| Ablation of cellular prion protein does not ameliorate abnormal neural network activity or cognitive dysfunction in the J20 line of human amyloid precursor protein transgenic mice M Cissé, PE Sanchez, DH Kim, K Ho, GQ Yu, L Mucke The Journal of Neuroscience 31 (29), 10427-10431, 2011 | 149 | 2011 |
| The C-terminal products of cellular prion protein processing, C1 and C2, exert distinct influence on p53-dependent staurosporine-induced caspase-3 activation C Sunyach, MA Cisse, CA da Costa, B Vincent, F Checler Journal of Biological Chemistry 282 (3), 1956-1963, 2007 | 97 | 2007 |
| Alzheimer's disease: A prion protein connection M Cisse, L Mucke Nature 457 (7233), 1090-1091, 2009 | 73 | 2009 |
| M1 and M3 muscarinic receptors control physiological processing of cellular prion by modulating ADAM17 phosphorylation and activity MA Cissé, C Sunyach, BE Slack, A Fisher, B Vincent, F Checler The Journal of neuroscience 27 (15), 4083-4092, 2007 | 65 | 2007 |
| Eph Receptors: New Players in Alzheimer’s Disease Pathogenesis M Cissé, F Checler Neurobiology of disease, 2014 | 48 | 2014 |
| Corrigendum to “ERK1-independent α-secretase cut of β-amyloid precursor protein via M1 muscarinic receptors and PKCα/ε”[Molecular and Cellular Neuroscience] 47 (2011) 223–232 M Cisse, U Braun, M Leitges, A Fisher, G Pages, F Checler, B Vincent Molecular and Cellular Neuroscience 48 (3), 266, 2011 | 46* | 2011 |
| ERK1-independent alpha-secretase cut of beta-amyloid precursor protein via M1 muscarinic receptors and PKC alpha/epsilon (vol 47, pg 223, 2011) M Cisse, U Braun, M Leitges, A Fisher, G Pages, F Checler, B Vincent MOLECULAR AND CELLULAR NEUROSCIENCE 48 (3), 266-266, 2011 | 46* | 2011 |
| ERK1-independent α-secretase cut of β-amyloid precursor protein via M1 muscarinic receptors and PKCα/ε M Cisse, U Braun, M Leitges, A Fisher, G Pages, F Checler, B Vincent Molecular and Cellular Neuroscience 47 (3), 223-232, 2011 | 46 | 2011 |
| Regulation of APP and PrPc cleavage by-secretase: mechanistic and therapeutic perspectives B Vincent, MA Cisse, C Sunyach, MV Guillot-Sestier, F Checler Current Alzheimer Research 5 (2), 202-211, 2008 | 46 | 2008 |
| The extracellular regulated kinase-1 (ERK1) controls regulated α-secretase-mediated processing, promoter transactivation, and mRNA levels of the cellular prion protein M Cissé, E Duplan, MV Guillot-Sestier, J Rumigny, C Bauer, G Pagès, ... Journal of Biological Chemistry 286 (33), 29192-29206, 2011 | 32 | 2011 |
| Design and characterization of a novel cellular prion-derived quenched fluorimetric substrate of α-secretase MA Cissé, C Gandreuil, JF Hernandez, J Martinez, F Checler, B Vincent Biochemical and biophysical research communications 347 (1), 254-260, 2006 | 29 | 2006 |
| The disintegrin ADAM9 indirectly contributes to the physiological processing of cellular prion by modulating ADAM10 activity. MA Cissé, C Sunyach, S Lefranc-Jullien, R Postina, B Vincent, F Checler Journal of Biological Chemistry 288 (32), 23433-23433, 2013 | | 2013 |
| The extracellular regulated kinase-1 (ERK1) controls regulated α-secretase-mediated processing, promoter transactivation, and mRNA levels of the cellular prion protein. M Cissé, E Duplan, MV Guillot-Sestier, J Rumigny, C Bauer, G Pagès, ... The Journal of biological chemistry 288 (27), 19646, 2013 | | 2013 |
| Methods of Treating Neurodegenerative Disease L Mucke, M Cisse US Patent App. 13/881,625, 2011 | | 2011 |
| Physiological Processing of the Cellular Prion Protein and βAPP: Enzymes and Regulation B Vincent, MA Cisse, F Checler Advances in Alzheimer’s and Parkinson’s Disease, 305-316, 2008 | | 2008 |
