Bruce Cronstein
Bruce Cronstein
NYU School of Medicine
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Adenosine receptors: therapeutic aspects for inflammatory and immune diseases
G Haskó, J Linden, B Cronstein, P Pacher
Nature reviews Drug discovery 7 (9), 759-770, 2008
Adenosine 5′-triphosphate and adenosine as endogenous signaling molecules in immunity and inflammation
MJL Bours, ELR Swennen, F Di Virgilio, BN Cronstein, PC Dagnelie
Pharmacology & therapeutics 112 (2), 358-404, 2006
Adenosine: an endogenous regulator of innate immunity
G Haskó, BN Cronstein
Trends in immunology 25 (1), 33-39, 2004
A mechanism for the antiinflammatory effects of corticosteroids: the glucocorticoid receptor regulates leukocyte adhesion to endothelial cells and expression of endothelial …
BN Cronstein, SC Kimmel, RI Levin, F Martiniuk, G Weissmann
Proceedings of the National Academy of Sciences 89 (21), 9991-9995, 1992
Adenosine, an endogenous anti-inflammatory agent
BN Cronstein
Journal of applied Physiology 76 (1), 5-13, 1994
The antiinflammatory mechanism of methotrexate. Increased adenosine release at inflamed sites diminishes leukocyte accumulation in an in vivo model of inflammation.
BN Cronstein, D Naime, E Ostad
The Journal of clinical investigation 92 (6), 2675-2682, 1993
Low-dose methotrexate: a mainstay in the treatment of rheumatoid arthritis
BN Cronstein
Pharmacological reviews 57 (2), 163-172, 2005
Adenosine: a physiological modulator of superoxide anion generation by human neutrophils.
BN Cronstein, SB Kramer, G Weissmann, R Hirschhorn
The Journal of experimental medicine 158 (4), 1160-1177, 1983
Adenosine: an endogenous inhibitor of neutrophil-mediated injury to endothelial cells.
BN Cronstein, RI Levin, J Belanoff, G Weissmann, R Hirschhorn
The Journal of clinical investigation 78 (3), 760-770, 1986
Molecular action of methotrexate in inflammatory diseases
ESL Chan, BN Cronstein
Arthritis Research & Therapy 4 (4), 1-8, 2002
Neutrophil adherence to endothelium is enhanced via adenosine A1 receptors and inhibited via adenosine A2 receptors.
BN Cronstein, RI Levin, M Philips, R Hirschhorn, SB Abramson, ...
The Journal of Immunology 148 (7), 2201-2206, 1992
The adenosine/neutrophil paradox resolved: human neutrophils possess both A1 and A2 receptors that promote chemotaxis and inhibit O2 generation, respectively.
BN Cronstein, L Daguma, D Nichols, AJ Hutchison, M Williams
The Journal of clinical investigation 85 (4), 1150-1157, 1990
Adenosine; a physiologic modulator of superoxide anion generation by human neutrophils. Adenosine acts via an A2 receptor on human neutrophils.
BN Cronstein, ED Rosenstein, SB Kramer, G Weissmann, R Hirschhorn
The Journal of Immunology 135 (2), 1366-1371, 1985
Understanding the mechanisms of action of methotrexate
H Tian, BN Cronstein
Bull NYU Hosp Jt Dis 65 (3), 168-173, 2007
Methotrexate inhibits neutrophil function by stimulating adenosine release from connective tissue cells.
BN Cronstein, MA Eberle, HE Gruber, RI Levin
Proceedings of the National Academy of Sciences 88 (6), 2441-2445, 1991
The adhesion molecules of inflammation
BN Cronstein, G Weissmann
Arthritis & Rheumatism 36 (2), 147-157, 1993
Methotrexate—how does it really work?
ESL Chan, BN Cronstein
Nature Reviews Rheumatology 6 (3), 175-178, 2010
Molecular therapeutics. Methotrexate and its mechanism of action
BN Cronstein
Arthritis & Rheumatism 39 (12), 1951-1960, 1996
Colchicine alters the quantitative and qualitative display of selectins on endothelial cells and neutrophils.
BN Cronstein, Y Molad, J Reibman, E Balakhane, RI Levin, G Weissmann
The Journal of clinical investigation 96 (2), 994-1002, 1995
The mechanism of action of methotrexate
BN Cronstein
Rheumatic disease clinics of North America 23 (4), 739-755, 1997
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